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Colon Cancer Prevention in Los Angeles
Posted January 27, 2010 by drtabib

celiac disease As we in our lives, it becomes more difficult to keep up with our health as we have to run around with responsibility. So by the time many of us realize that our health is slipping it may be too late. And what if you could do something in order to achieve a form of colon cancer prevention?

A common form of colon cancer diagonosist is in the colon polyps. An experienced gastric doctor would be able to diagnose you with the aid if a camera guided scope. He or she will be able to pin point an almost precise diagnosis in order to better treat your condition.

The main symptom of gastroesophageal reflux disease for adults is frequent heartburn, also called acid indigestion, a burning pain in the mid-chest, behind the breast bone and sometimes in the middle abdomen. Some children under 12 years may suffer from reflux disease without heartburn. These can sometimes be diagnosed with sensations of dry cough, asthma like symptoms or trouble swallowing. If your child be suffering form similar symptoms you may want to consult a gastro specialist doctor about what options you may have.

Ever since the discovery of Celiac disease doctors have been working towards ways of treating this hereditary disease in order to help those suffering with this to live better lives. It must be very difficult to live with out being able to eat breads, pastas and other certain foods in fear that you may not be able to digest the food or suffer discomfort. Seeking the help of a qualified specialist can offer you several options to help you deal with celiac disease, and offer ways to help control it. Talk to board certified gastric doctor about what options you may have.

drtabib
A new study reported by Nutrition Data suggests that Celiac disease is four times more common today than it was even fifty years ago. That's an extraordinary finding. And lest we think that this is simply due to an uptick in awareness and diagnosis, the researchers disproved this with a very interesting approach. They tested stored blood samples taken from American soldiers in the 1950s. Only one in every 700 samples contained the antibody that signals celiac disease. Today, about one in 170 people test positive for the antibody.

gastroesophageal reflux diseaseCeliac is characterized by an intolerance to gluten, a protein found in wheat and other cereal grains. When challenged with gluten, the body's immune system attacks the cells lining the small intestine causing digestive chaos and nutrient malabsorption. It's treated by strict avoidance of gluten-containing products. This gastrointestinal disease is different from another common disease called gastroesophageal reflux disease that affects the esophageal and stomach areas of the digestive system.

With regards to the increase in numbers of Celiac disease cases, scientists are stumped. But it strikes me that we might consume a lot more gluten today then they did in the 50s. As anyone trying to avoid gluten can tell you, gluten is in virtually every type of processed food and our diet has become increasingly dominated by processed foods. Perhaps over-exposure to this protein increases the chance of developing an intolerance?

Other possible explanations include environmental and lifestyle changes. As people spend more time indoors (and slather themselves with sunscreen when they go out), vitamin D deficiency has been on the rise, and has been linked with other auto-immune conditions, particularly multiple sclerosis.

Perhaps widespread use of over-the-counter and prescription acid-blockers (unavailable in the 50s) plays a role in this digestive disorder. All that stomach acid we're suppressing normally helps break down proteins. Incompletely digested proteins could be more likely to provoke an immune reaction, especially if they "leak" into the blood stream from the large intestine.

Perhaps a decrease in our consumption of naturally-fermented foods with beneficial bacteria plays a role? Beneficial bacteria help maintain the integrity of the intestinal lining and prevent large molecules (such as intact proteins) from being absorbed into the bloodstream.

It doesn't seem like a stretch to imagine that the increase in refined sugar consumption could be related. A high sugar diet can affect the intestinal flora and function as well as determine whether or not an individual develops colon polyps. There are a lot of paths for future inquiry.

The good news is that manufacturers are responding to the growing demand for gluten free products. Avoiding gluten has become fashionable among people without celiac disease as well. A return to unprocessed, whole foods can also make gluten avoidance easier--no mysterious ingredients, no hidden gluten. For more information on Celiac disease and colon cancer prevention contact your local Gastroenterology and Hepatology specialists.

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An American Association for Cancer Research news release this month reports that a genetic mutation may explain why blacks are more likely than whites to have a more aggressive form of colorectal cancer. "Several studies have identified a disparity between African-Americans and whites for colorectal cancer. What this study does is pinpoint a possible genetic cause," study author Upender Manne, an associate professor in the pathology and Los Angeles colon cancer prevention department at the University of Alabama at Birmingham, said in the news release.

They analyzed DNA from 137 black colorectal cancer patients and 236 white patients, and found that both groups had similar rates of p53 mutations. However, the frequency of Pro72 allele of p53 was found in 17% of blacks and in only 7% of whites. The Arg72 allele was found in 36% of whites, and in 19% of blacks. The study intentionally did not account for non-cancer related gastrointestinal conditions such as Beverly Hills gastroesophageal reflux disease (commonly known as GERD) when pursuing its findings.

The presence of Pro72 allele in blacks was associated with a more than twofold increase in death due to colorectal cancer, the researchers found. "This paper shows that in a subset of patients with the Pro72 allele, the susceptibility to p53 mutations may be a possible molecular explanation for the racial disparity," Manne said. The study appears in the current April 2009 issue of Clinical Cancer Research.

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Learning more about the relationship between molecular defects and differences in colorectal cancer incidence, aggressiveness and patient outcomes may help lead to individualized treatments and the elimination of racial disparities like the development of Beverly Hills colon polyps, the researchers wrote.

The discovery boosts the scientific understanding of racial disparities in cancer and other diseases like Los Angeles Celiac disease and adds new detail in the ongoing search for more personalized cancer-fighting therapies, said Upender Manne, Ph.D., an associate professor in the UAB Department of Pathology who led the study.

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